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The Host — Autistic Neurology and the Tensor Tympani
The worker is awaiting formal autism diagnosis following self-identification and assessment
referral. For thirty years prior to this injury, he had built and maintained an extraordinarily
effective set of coping mechanisms—the kind that allow a high-functioning autistic person to
work mainline railway track in blizzards, to calibrate acoustic environments with precision,
and to never miss a shift.
The acoustic injury on January 12, 2026, did not simply affect a generic auditory system. It
attacked the specific, finely tuned sensory architecture of an autistic nervous system. Autistic
sensory processing operates with higher gain and lower adaptive threshold for repetitive,
tonal, sustained stimuli than neurotypical processing. A sustained 328 Hz mechanical
resonance, cycling continuously for ten-hour days across eleven days, is precisely the
category of stimulus that an autistic nervous system cannot habituate to, cannot filter, and
cannot recover from while re-exposure continues.
The clinical outcome was Tonic Tensor Tympani Syndrome—TTTS. The tensor tympani is a
small muscle inside the middle ear whose function is to dampen intense sounds. Sustained
acoustic irritation, particularly at frequencies that resonate with the tensor tympani's own
mechanical response profile, can drive the muscle into sustained, involuntary spasm. The
symptoms produced are exact: unilateral ear pain (left), aural fullness, tinnitus worsening,
sensitivity to sound, and the cascade of autonomic dysregulation—disrupted sleep,
heightened startle response, anxiety, and exhaustion—that follows when the middle ear can
no longer regulate its own mechanical gain.
This was not a migraine. The worker had managed migraines successfully for years without
a single sick day. What TTTS produces is categorically different from migraine in its
mechanism, its symptom profile, and its response to treatment. The distinction matters
because the Clinical Opinion that eventually denied this claim would collapse these two
distinct clinical realities into one convenient label and call the result pre-existing.
ACT Il: THE ADMINISTRATIVE FRICTION AND THE
AUDIT
The Clinical Opinion — What It Said and What It Ignored
On March 20, 2026, WorkSafeBC Audiologist Advisor Flora Pang, M.Sc., RAUD, Aud (C),
issued a five-page Clinical Opinion concluding that the incident was "not consistent with
acoustic trauma" and that "the main driving factors of the worker's complaints are likely his
pre-existing and non-occupational conditions of migraines and noise sensitivity, rather than
the incident itself."
In reaching this conclusion, the Clinical Opinion made no reference to the 328 Hz resonance
data. It made no reference to Tonic Tensor Tympani Syndrome. It characterized the worker's
heightened acoustic sensitivity—a direct feature of his autistic neurological profile—as a
generic "pre-existing condition.” And it deployed, as its central environmental safety
argument, a comparison between the OMC1 Training Room and "a BC Ferry data center
and a central computer processing area of a large hospital."